| Author | Message | ||
     RANDALL TILLMAN New member Username: rtillman Post Number: 11 Registered: 06-2006 Posted From: 206.197.1.3 Rating: N/A |
LAST NIGHT GAVE HER 30CC OF NUTRA DRENCH, 2CC BANAMINE, 2CC THIAMINE, AND 5CC B12 COMPLEX. DOESN'T LOOK LIKE SHE'S MAKING A WHOLE LOT OF PROGRESS, BUT SHE'S STILL HANGING IN THERE. HER EYES ARE SQUINTED TOGETHER,AND SHE RUNS INTO THE WALL. SHE STILL KEEPS HER HEAD TURNED WITH HER NOSE TOUCHING HER SIDE. NOT EATING THAT I KNOW OF, OR DRINKING.BUT HER BODY STILL LOOKS PRETTY HEALTHY.WILL LET YOU ALL KNOW IF THERE ARE ANY CHANGES. THANKS FOR ALL YOUR HELP!!! SINCERLEY RANDALL TILLMAN P.S. SHE LOOKED LIKE SHE HAD SOME SORT OF FECAL MATTER IN HER MOUTH. | ||
| Elaine Elder New member Username: elaine Post Number: 62 Registered: 12-2006 Posted From: 67.34.29.175 Rating: N/A |
Also Polioencephalomalacia Use same Vitamin B complex Therapy as Deer worm and use Anti Inflamitory Pathology: Polioencephalomalacia (PEM), also known as cerebrocortical necrosis, is a disease characterized by a disturbance of the central nervous system. The brain of infected animals becomes inflamed and swollen, and eventually becomes necrotic. Diagnosis is usually done by performing a necropsy on the brain of the dead animal. Dead gray matter will fluoresce under a Wood's lamp. Causes: PEM sometimes occurs on high grain diets, and diets that include plants high on thiaminases and sulfur. Thiaminases are enzymes found in a few plants, such as bracken fern, and the raw flesh and viscera of certain fish and shellfish. When ingested these enzymes split thiamin (Vitamin B1), an important compound in energy metabolism, and render it inactive. Normally ruminants are fairly resistant to thiamin deficiency since rumen microbes provide the animal with sufficient amounts of thiamin. However, the ingestion of thiaminases will lead to deficiency. Additionally, young growing ruminants, especially cattle and sheep, fed high-grain diets are especially susceptible. Diets high in grains can encourage the growth of certain thiaminase-producing bacteria in the rumen. These bacteria, including Clostridium sporogenes and a few species of Bascillus can produce enough thiaminases to induce thiamin deficiency. A thiamine-analogue is also produced within the rumen if there is excess sulfur, which may replace thiamine in important metabolic reactions in the brain. When thiamine is deficient, key tissues that require large amounts of thiamine, such as the brain and heart, are the first to show lesions. Clinical signs: This usually occurs suddenly. Affected sheep stand or sit alone, are blind and arch their necks back and stare upwards and become "star gazers", the medical name for this being opishotonus. They are disoriented, lose their appetite, and they do not want to drink. Temperature and respiratory rate are usually normal but the heart rate may be depressed. Excitement may be seen but is usually replaced with dullness. Normally only a few individuals are affected. The animal may go down on its side with its head thrown back. The legs may be rigidly extended and convulsions may occur. Animals with PEM will often press their head against a wall or post. If not treated on time, most animals with PEM will die within 48 hours. Treatment and prevention: Sheep suffering from polioencephalomalacia generally respond very well to treatment if caught early. They can be successfully treated with 200 to 500 mg of thiamin injected intravenously, intramuscularly, or subcutaneously. Because thiamine is water-soluble, it is quickly eliminated from the body through the kidneys and, therefore there is little risk of overdosing. Dexamethasone is often administered along with thiamine to reduce brain swelling. Although recovery is usually quick, if significant brain damage has occurred, the recovered sheep rarely regain satisfactory levels of productivity. Therefore, very early treatment is critical. If a case of PEM is diagnosed in a herd of sheep, it is advisable to inject the remaining animals with thiamine as prevention. Drinking water should be tested for sulfur contents, sources of thiaminases, if any, should be removed and animals should be introduced to grain diets in steps to avoid a sudden increase in thiaminases-producing bacteria in the rumen. References: Sheep Production Handbook, Oct. 1988. Sheep Industry Development Program, INC. Pg 7-8. http://www.inform.umd.edu/EdRes/Topic/AgrEnv/ndd/goat/METABOLIC_AND_NUTRITIONAL_ DISEASES.html http://www.ansci.cornell.edu/plants/toxicagents/ | ||
| Elaine Elder New member Username: elaine Post Number: 61 Registered: 12-2006 Posted From: 67.34.29.175 Rating: N/A |
I have been doing some web research. A hundred years ago, the U.S. deer population was a sparse 500,000. Today, experts estimate that 20 million deer roam the nation – in both rural and suburban settings. “In the last decade, from the Rockies to New England and the Deep South, rural and suburban areas have been beset by white-tailed deer gnawing shrubbery and crops, spreading disease and causing hundreds of thousands of auto wrecks,” The New York Times reported “Fast-multiplying herds are altering the ecology of forests, stripping them of native vegetation and eliminating niches for other wildlife.” Treatment of meningeal worm infection is most successful when instituted early in the course of disease. A treatment regime which has proven successful at the Ohio State University involves fenbendazole (20 to 50 mg/kg body weight, PO, q24h for 5 days) and flunixin meglumine (1 mg/kg, IV, IM, or SC, q12h for 5 days) or dexamethasone in non-pregnant females and males (0.1 mg/kg, IV, IM, or SC, q24h for 3 days). DMSO (1g/kg given in 500 ml of 5% dextrose solution, IV, q24h) given to effect is useful in some cases but may cause severe appetite suppression. Discontinue DMSO if inappetance or anorexia occurs. Vitamin E, selenium, Vitamins B-complex, and Vitamin A are useful to assist healing of neural tissues. antiinflammatory drugs are critical to reduce the inflammation associated with the presence of the migrating larvae and the subsequent inflammatory response to the killed larvae. Use of antiinflammatory drugs is important to prevent the clinical signs from becoming more severe after instituting treatment. See this website for more Information http://goatconnection.com/articles/publish/article_126.shtml Hope this helps | ||
| Elaine Elder New member Username: elaine Post Number: 60 Registered: 12-2006 Posted From: 67.34.29.175 Rating: N/A |
The menengeal worm or white tailed deer worm is a gastro intestinal parasite in deer only. The infected deer travel through your pasture, leaving infected droppings in dense underbrush where a tiny slug that likes damp places comes into contact with the feces. The slug lives on the under side of damp leaves. A goat comes along and eats the leaf and accidentally ingests the slug with it. the slug is only a quarter to a half inch long. The slug is digested but the parasite is released into the stomach. In goats, llamas and alpacas it is not in the correct host so it gets into the blood stream, wandering around looking for where it is supposed to be. Oral parasite control only works in the digestive system. it does not cross into the blood stream. To control the deer parasite in the blood stream the wormer has to be given by injection. The symptoms of menengeal parasite are hard to recognize before the parasite makes its way to the spinal cord, where it causes inflamation, which causes stroke like symptoms. Holding the head in an odd position was the first symptom I noticed. Dragging back feet was next. Not being able to walk in a straight line, They are looking one direction and their body goes a different way, they stop, turn toward their objective, and still can't make their body go where they want. Eventually, the head curves around and touches the side and just stays that way. After that they lose mobility. The whole body curls up. If you try to straighten them out they flail about like a fish out of water. I have had four goats get this. 2 lived, 2 did not. Since then, I have cleared the dead leaves and dense underbrush, to make it less hospitible to the slug. Usually after a few cold weeks kill off the slugs the danger of goats catching the worm go down until the next spring. None of mine lost any hair or wool. (Angora) The parasite can live in the blood stream for up to 30 days. Sometimes never making it to the spine. So an entire herd could be infected but only one or two have symptoms. All four of mine had their head curled around just be fore they lost total mobility. It takes months for them to recover from the final stages. The two I lost died the first day I found them all curled up. My big angora has just learned how to run again. Just not in a straight line. She has a zombie like gait and holds her head tilted to the side. It has been over a year since she got it. It took 6 months for her to be able to walk on her own. If your goat doesn't have any of these symptoms I would look at other options. giving ivermectin by injection does more for controlling the deerworm than it would if given orally. Use 3x the cattle dosage SQ. Also give Vitamin B complex If it is this one - COMPOSITON: Each mL of sterile aqueous solution contains: Thiamine Hydrochloride (B1) 100 mg you can give 1 cc per 20 pounds every 4 to 6 hours for the first 24 hours then 2 to 3 times a day for 5 to 7 more days.. If it is this one - COMPOSITION: Each mL of sterile aqueous solution contains: Thiamine Hydrochloride (B1) 12.5 mg you need to give 1 cc per 3 pounds every 4 to 6 hours for the first 24 hours then 2 to 3 times a day for 5 to 7 more days. Just as a precaution if you have other goats, If you think this one has the deer worm, Go ahead and give them an injection of Ivomec, in case they have the parasite but no symptoms. | ||
| RANDALL TILLMAN New member Username: rtillman Post Number: 10 Registered: 06-2006 Posted From: 65.7.65.130 Rating: N/A |
SHE WEIGHS ABOUT 85 TO 90 LBS.CHECKED HER TEMP TONIGHT 100 F DEGREES.HER RUNNY NOSE LOOKS YELLOWISH TO LIGHT BROWN. I AM KIND NEW TO THE GOAT BUSINESS,HAVEN'T TRIED FEELING OF RUMEN YET. SHE KEEPS HER HEAD TURNED TO HER RIGHT SIDE STANDING UP,IF SHE'S NOT DOING THAT HER HEAD IS HANGING DOWN, OR SHE HAS HER HEAD IN A CORNER. AS FOR THE REST OF THE STUFF YOU MENTIONED I WILL CHECK TOMORROW. THANKS FOR ALL OF YOUR HELP!!! | ||
| Barbara Howard Advanced Member Username: bhoward Post Number: 180 Registered: 08-2005 Posted From: 206.157.148.202 Rating: N/A |
First what will she weigh? What is her temp? What color is her runny nose. Have you felt for ruminant movement yet or have you seen her cudding? Is the head back toward her side lying down? Any droolinf or rapid eye movement? What color are the inner eyelids and gums? Get back to me ASAP on this email me personally if you want jamesa@mrtc.com Barbara Howard Capri-Medic jamesa@mrtc.com 606-522-3388 KY | ||
| (Unregistered Guest) Unregistered guest Posted From: 206.197.1.3 Rating: N/A |
SHE'S STILL ALIVE,BUT SHE'S NOT VERY ACTIVE. SHE STANDS AROUND WITH HER HEAD BENT BACK TOUCHING HER SIDE. I HAVE BEEN GIVING HER 3CC OF NUFLOR, 5CC B12 COMPLEX,AND STARTED LAST NIGHT GIVING 20CC OF NUTRA DRENCH. DOES ANYBODY KNOW WHAT MAY BE WRONG WITH HER. | ||
| Elaine Elder New member Username: elaine Post Number: 57 Registered: 12-2006 Posted From: 216.9.250.6 Rating: N/A |
When ever they go off feed or when you give antibiotics it disrupts the digestive process. I would get probios, or something from feed store for horses that puts digestive enzymes back into system. If not available, get some yougurt with active enzymes. I would keep up the vitamin B. | ||
| Gary Pfalzbot Board Administrator Username: admin Post Number: 106 Registered: 07-2005 Posted From: 67.142.130.27  Rating: N/A |
Randall, I'm not sure of any contraindications between LA 200 and Nuflor but you might want to wait at 24 hours or so before you give her the Nuflor. The fact that she's eating is a good sign - it's usually progressed pretty far when they won't eat, won't drink. As long as she is eating, I'd offer her some slices of orange, peel and all, and pretty of water...you could even add some molasses or use the MAGIC formula... Then in the morning go ahead and give the Nuflor and watch her carefully from there. Sounds like you may have caught this in time so there is a good chance you'll be able to save her. Please keep us posted. Best regards, GP Gary Pfalzbot, GoatWorld | ||
| RANDALL TILLMAN New member Username: rtillman Post Number: 9 Registered: 06-2006 Posted From: 65.7.64.210 Rating: N/A |
I HAVE A YOUNG NANNY ABOUT ONE YEAR OLD SHE HAS BAD RUNNY NOSE, BREATHING HARD, SOUNDS LIKE SHES RATTLING IN HER CHEST. I HAVE GAVE HER 4CC OF LA 200,5CC OF B-12,AND 150 CC OF ELECTROLYTES SHES LAYING DOWN PRETTY WEAK I DON'T THINK SHE CAN GET UP. HER HEAD WAS DOWN, BUT NOW SHE 'S HOLDING HER HEAD AND EATING A LITTLE HAY. DO YOU THINK I CAN GIVE HER SOME NUFLOR SINCE I ALEADY GAVE LA 200. PLEASE LET ME KNOW IF THERE IS ANYTHING I COULD DO FOR. P.S. I ALSO HAVE HER LAYING UNDER A HEAT LAMP. |